つくばリポジトリ (Tulips-R) > 160 人間総合科学研究科 (Graduate School of Comprehensive Human Sciences) > 18 疾患制御医学専攻 (Clinical Sciences) >
A Novel Diabetes Mellitus Mouse Model, MAFA-Deficient and Beta Cell-Specific MAFK-Overexpressing Hybrid Transgenic Mice, Developed Severe Diabetic Nephropathy and Improved with TCV-116 (Candesartan Cilexetil) Treatment
MafA-deficient and beta cell-specific MafK-overexpressing hybrid transgenic mice develop human-like severe diabetic nephropathy
Nrf2 regulates the sensitivity of death receptor signals by affecting intracellular glutathione levels
